Papers

Generation of an endogenous auxin inducible degron-tagged SPAS-1/spastin to investigate its targeted depletion in C. elegans neurons

Brown, E., Kuszynski, S., Akoachere, F., Feduccia, J., Malatinszky, L., & Luth, E. S. (2024). microPublication biology, 2024, 10-17912.

Sublethal perfluorooctanoic acid and perfluorooctanesulfonic acid delay C. elegans larval development and population growth but do not alter egg hatching

Breton, C., Kessel, K., Robinson, A., Altaf, K., & Luth, E. S. (2024). Journal of Toxicology and Environmental Health, Part A, 87(1), 22-32.

A Versatile Semester-Long Course-Based Undergraduate Research Experience using Optogenetics and RNAi to Identify Genes Important for Synapse Function

Luth, E.S.. & Juo, P. (2023). Journal of Neuroscience Education, 22(1), A1-A13.

Regulation of AMPA receptor trafficking by secreted protein factors

Rennich, B. J., Luth, E. S., Moores, S., & Juo, P. (2023).. Frontiers in Cellular Neuroscience, 17, 1271169.

DMD-10 is dispensable for the initial development of amphid sensory neurons and their survival in mature C. elegans

Nguyen, I., & Luth, E. S. (2021). microPublication Biology, 2021, 10-17912.

The Doublesex/Mab-3 domain transcription factor DMD-10 regulates ASH-dependent behavioral responses

Durbeck, J., Breton, C., Suter, M., Luth, E. S., & McGehee, A. M. (2021). PeerJ, 9, e10892.

VER/VEGF receptors regulate AMPA receptor surface levels and glutamatergic behavior

Luth, E. S., Hodul, M., Rennich, B. J., Riccio, C., Hofer, J., Markoja, K., & Juo, P. (2021). PLoS Genetics, 17(2), e1009375.

The Snail transcription factor CES-1 regulates glutamatergic behavior in C. elegans

Park, L., Luth, E. S., Jones, K., Hofer, J., Nguyen, I., Watters, K. E., & Juo, P. (2021). Plos one, 16(2), e0245587.

The WD40-Repeat Protein WDR-20 and the Deubiquitinating Enzyme USP-46 Promote Cell Surface Levels of Glutamate Receptors

Hodul, M., Rennich, B. J., Luth, E. S., Dahlberg, C. L., & Juo, P. (2021). Journal of Neuroscience, 41(14), 3082-3093.

Measuring Mitochondrial Dysfunction Caused by Soluble α-Synuclein Oligomers

Luth, E. S., & Stavrovskaya, I. G. (2019). Methods in molecular biology (Clifton, NJ), 1948, 183-198.

Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiation

Dettmer, U., Newman, A. J., Soldner, F., Luth, E. S., Kim, N. C., Von Saucken, V. E., ... & Selkoe, D. (2015). Nature communications, 6(1), 7314.

Purification of α-synuclein from human brain reveals an instability of endogenous multimers as the protein approaches purity

Luth, E. S., Bartels, T., Dettmer, U., Kim, N. C., & Selkoe, D. J. (2014). Biochemistry, 54(2), 279.

Soluble, prefibrillar α-synuclein oligomers promote complex I-dependent, Ca2+-induced mitochondrial dysfunction

Luth, E. S., Stavrovskaya, I. G., Bartels, T., Kristal, B. S., & Selkoe, D. J. (2014). The Journal of biological chemistry, 289(31), 21490-21507.

Bone marrow side population cells are enriched for progenitors capable of myogenic differentiation

Luth, E. S., Jun, S. J., Wessen, M. K., Liadaki, K., Gussoni, E., & Kunkel, L. M. (2008). Journal of cell science, 121(Pt 9), 1426-1434.